Compound Profile

CJC 1295

GHRH analog peptide

CJC 1295 is a synthetic growth hormone releasing hormone (GHRH) analog engineered for a longer half life than natural GHRH, commonly combined with Ipamorelin in gray market protocols; its GH/IGF 1 mechanism has early pharmaceutical development data behind it, but no FDA approval and an uncertain compounding pathway.

Last reviewed:

C Preliminary

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CJC 1295

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Unapproved compound: FDA compounding status uncertain

CJC-1295 is not FDA-approved for any human use. It was removed from the FDA’s Category 2 restricted 503A bulk substances list in April 2026 following withdrawal of its nomination, but was not moved to the approved Category 1 list and is not on the July 2026 FDA Pharmacy Compounding Advisory Committee (PCAC) review agenda.

  • Research-use-only (RUO) product is not legally intended for human consumption.
  • No long-term human safety or efficacy data exists for any dose or protocol.
  • This profile is not a self-treatment guide.

Regulatory status

2026 federal and state regulatory context

Last checked 2026 06 30

Sellable only as research use only (RUO) product; compounding pharmacy pathway is uncertain and not scheduled for near term PCAC review.

Regulatory changelog

  • 2023 04/2026: FDA Category 2 restricted (503A bulk substances). 2026 04: Removed from Category 2 following nomination withdrawal (gray zone, not Category 1). Not included on the July 23 24, 2026 PCAC review agenda (which covers BPC 157, KPV, TB 500, MOTS C, Emideltide/DSIP, Semax, and Epitalon).

Quick Stats

Evidence level

Preliminary evidence

Typical onset

Varies by prep

Safety rating

Use extra caution: Safety review pending

Best for

GHRH Receptor Agonism, DAC Albumin Binding Half Life Extension, Downstream IGF 1 Elevation

Avoid / review if

Pregnancy/Breastfeeding, Uncontrolled Diabetes, Unsupervised RUO Product Use Without Physician Oversight

Safety & Cautions

Review before use if any apply: Pregnancy/Breastfeeding, Uncontrolled Diabetes, Unsupervised RUO Product Use Without Physician Oversight.

Evidence-based safety

Caution when combined

These pairings share a flagged risk mechanism. They are additive-effect cautions derived from contraindication data, not confirmed clinical interactions. Consult a clinician before combining.

Evidence Summary

C Preliminary

Evidence lens

Early signal that needs stronger human replication before practical claims.

PreliminaryPreliminary evidence

Human clinical evidence: Not the primary signal

Mechanistic / preclinical: Mechanism mappedGHRH Receptor Agonism · DAC Albumin Binding Half Life Extension · Downstream IGF 1 Elevation

Research maturity: Preliminary or mixedmain contexts: GHRH Receptor Agonism · DAC Albumin Binding Half Life Extension · Downstream IGF 1 Elevation

Safety boundary: Safety note availableReview before use if any apply: Pregnancy/Breastfeeding, Uncontrolled Diabetes, Unsupervised RUO Product Use Without Physician Oversight.

Evidence Strength

Confidence estimate based on the design quality and consistency of published clinical trials.

Preliminary evidence

CJC 1295 has a preliminary evidence evidence rating.

General wellnessGeneral wellness
Effects
3
How evidence grades work

Each grade reflects the strength and consistency of published human evidence — not marketing claims. Grades are based on study count, design quality, effect size, consistency, and recency.

A

Strong

Multiple RCTs, consistent direction, adequate effect size

B

Moderate

Some RCTs or consistent observational data in humans

C

Preliminary / Mixed

Animal or in-vitro only, or conflicting human data

D

Traditional / Theoretical

Traditional use only; no controlled human trials

How CJC 1295 Works

Simplified mechanism pathway based on preclinical and pharmacological evidence. Does not confirm clinical efficacy.

Mechanism Pathway — How CJC 1295 Works

How CJC 1295 WorksCJC 1295 acts on pathways via ghrh receptor (agonism), leading to ghrh receptor agonism.CJC 1295Biological pathwayGHRH ReceptorAgonismGhrh Receptor AgonismObservable outcomeDAC Albumin BindingHalf Life Extension
CompoundTarget / ReceptorMechanismEffect / Outcome
Mechanisms & Biological Pathways

Preclinical mechanism details from scientific profiles; these represent plausible pathways but do not guarantee clinical efficacy in humans.

GHRH Receptor AgonismDAC Albumin Binding Half Life ExtensionDownstream IGF 1 ElevationGrowth Hormone AxisIGF 1 Signaling

Compare & Sourcing

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⚠️ Sourcing Options Disabled for Safety

Direct product recommendations and affiliate links are suppressed for this compound due to its high caution or needs-review safety classification.

Evaluate the safety checks, contraindications, and potential medication interactions below under clinician supervision before use.

Safety first · Harm reduction

This information is educational and not medical advice. Start low, avoid risky combinations, and consult a licensed clinician before making health decisions—especially if you use medications, have diagnosed conditions, or are pregnant/breastfeeding.

Learn how we evaluate confidence, safety, and intensity on the methodology page.

Editorial Review

Checked against primary sources, cited evidence, and contraindication language before publication. Evidence claims, safety language, and affiliate modules are reviewed independently. Not personal medical advice.

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Compound profile context

How to interpret CJC 1295

CJC 1295 dosage by use case, onset and duration, safety limits, and interactions for ghrh receptor agonism, graded against research. Mechanistic Evidence… Use this compound profile to understand mechanism, evidence quality, and practical safety context before comparing products or building a stack. A biochemical pathway connection can be useful for discovery, but it is not the same as proven benefit in humans.

When reviewing CJC 1295, separate the active molecule from the product category around it. Dose form, absorption, extract standardization, medication interactions, and individual sensitivity can change the real-world risk-benefit picture.

For supplement planning, avoid combining multiple compounds with overlapping sedating, stimulating, serotonergic, blood-pressure, anticoagulant, or liver-metabolism effects unless a qualified clinician has reviewed the context.